| KennethBus | Дата: Суббота, 11.10.2014, 23:04 | Сообщение # 1 |
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Статус:  | With no readily obtainable drug to save Alzheimer's illness (AD)—and nobody on the immediate scope—researchers are searching for potential causes and contributors to the most common neurodegenerative disorder.
An article published onwards of language in Neurochemistry International this week discussed epigenetic mechanisms that be experiencing been linked to AD. These mechanisms modulate gene pronouncement patterns using DNA methylation, histone post-translational modifications, or microRNAs, but they consign the entire DNA cycle unaltered.
Every cell in an specific has the verbatim at the same time DNA chain, but epigenetic fixing occurs at definitive gene loci in sure cells to sire precise cellular phenotypes. Humans gain these mutations as they behoove exposed to environmental factors such as victuals and toxins throughout their lives. When epigenetic alterations come to pass, genetic transcriptional movement may mutate and contrive risk after individual diseases, especially neurodegenerative disorders like AD.
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